MARQ 0.0.1

data/install_scripts/GEO/series/GSE8326.yaml

@@ -0,0 +1,37 @@
+--- 
+:description: |-
+  Yeast is a powerful model system for studying the action of small molecule therapeutics. An important limitation has been low efficacy of many small molecules in yeast due to limited intracellular drug accumulation. We used the DNA binding domain of the pleiotropic drug resistance regulator Pdr1 fused in-frame to transcription repressors to repress Pdr1 regulated genes. Expression of these regulators conferred dominant enhancement of drug sensitivity and led to greatly diminished levels of Pdr1p regulated transcripts, including the yeast p-glycoprotein homologue Pdr5. Enhanced sensitivity was seen for a wide range of small molecules. Biochemical measurements demonstrated enhanced accumulation of rhodamine in yeast cells carrying the chimeras. These repressors of Pdr1p regulated transcripts can be introduced into large collections of strains such as the S. cerevisiae deletion set, and enhance the utility of yeast for studying drug action and for mechanism-based drug discovery.
+  Keywords: Comparison of genetic variants
+:arrays: 
+  GSM206389: 
+    condition: Dpdr1
+  GSM206390: 
+    condition: Pdr1-Cyc8 fusion Dpdr1
+  GSM206391: 
+    condition: Pdr1-Cyc8 fusion Dpdr1
+  GSM206392: 
+    condition: Pdr1-Cyc8 fusion Dpdr1
+  GSM206381: 
+    condition: Empty vector
+  GSM206393: 
+    condition: Pdr1-Sin3
+  GSM206382: 
+    condition: Empty vector
+  GSM206394: 
+    condition: Pdr1-Sin3
+  GSM206383: 
+    condition: Empty vector
+  GSM206395: 
+    condition: Pdr1-Sin3
+  GSM206384: 
+    condition: Pdr1-Cyc8
+  GSM206385: 
+    condition: Pdr1-Cyc8
+  GSM206386: 
+    condition: Pdr1-Cyc8
+  GSM206387: 
+    condition: Dpdr1
+  GSM206388: 
+    condition: Dpdr1
+:title: Repression of pleiotrophic drug resistance genes in Saccharomyces cerevisiae using chimeric transcriptional repressors
+:platform: GPL90

data/install_scripts/GEO/series/GSE8399.yaml

@@ -0,0 +1,13 @@
+--- 
+:description: |-
+  The Saccharomyces cerevisiae Zn(II)2Cys6 zinc cluster transcription factor War1, mediates part of the transcriptional response to weak organic acids such as sorbate or benzoate.  We identified gain-of function alleles of War1 that map to a region in the center of the molecule conserved in War1 related proteins from other fungi. Independent of weak acids, they increase levels of plasma membrane ABC transporter Pdr12, the main target gene of War1. In the experiment, the constitutive War1 allele K762R was expressed from the GAL1 promoter. RNA was extracted after one hour induction of the GAL1 promoter by Galactose to exclude indirect effects of the constitutive allele.
+  Keywords: mutant vs wild type
+:arrays: 
+  GSM207891: 
+    condition: War1-C
+  GSM210125: 
+    condition: War1-C
+  GSM210126: 
+    condition: War1-C
+:title: WAR1-C mutants
+:platform: GPL3458

data/install_scripts/GEO/series/GSE850.yaml

@@ -0,0 +1,15 @@
+--- 
+:title: Effect 14-3-3 mutation on mRNA levels in yeast.
+:description: |-
+  Studies on the effect of a bmh2 mutation on the genome-wide steady-state mRNA levels in Saccharomyces cerevisiae.
+  Keywords: other
+:arrays: 
+  GSM13009: 
+    condition: CEN-PK113-7DvsCEN-PK113-7D
+  GSM13010: 
+    condition: GG3096 vs CEN-PK113-7D
+  GSM13011: 
+    condition: GG3096 vs CEN-PK113-7D
+  GSM13012: 
+    condition: GG3096 vs CEN-PK113-7D
+:platform: GPL583

data/install_scripts/GEO/series/GSE8506.yaml

@@ -0,0 +1,32 @@
+--- 
+:description: |-
+  Transription profile of Saccharomyces cerevisiae SK1 cultures undergoing synchronous sporulation.
+  We have measured mRNA levels in synchronized SK1 cells immediately upon transfer to the sporulation medium and every 30 minutes after that for 6 hours.  mRNA extracted from these cultures were converted to cDNA and hybridized to microarrays and log2 ratios of hybridization signal of each time point was compared to that of time zero (immediately prior to transfer to the sporulation medium).
+  Keywords: Time course
+:arrays: 
+  GSM211001: 
+    condition: sporulation_3.5hr
+  GSM211002: 
+    condition: sporulation_4.0hr
+  GSM211003: 
+    condition: sporulation_4.5hr
+  GSM211004: 
+    condition: sporulation_5.0hr
+  GSM211005: 
+    condition: sporulation_5.5hr
+  GSM211006: 
+    condition: sporulation_6.0hr
+  GSM210995: 
+    condition: sporulation_0.0hr
+  GSM210996: 
+    condition: sporulation_0.5hr
+  GSM210997: 
+    condition: sporulation_1.0hr
+  GSM210998: 
+    condition: sporulation_1.5hr
+  GSM211000: 
+    condition: sporulation_2.5hr
+  GSM210999: 
+    condition: sporulation_2.0hr
+:title: Yeast (S. cerevisiae SK1) Sporulation/Meiosis
+:platform: GPL4625

data/install_scripts/GEO/series/GSE8542.yaml

@@ -0,0 +1,47 @@
+--- 
+:description: |-
+  Cells in glucose-limited Saccharomyces cerevisiae cultures differentiate into quiescent (Q) and non-quiescent (NQ) fractions prior to entering stationary phase.  To identify genes involved in this differentiation, Q and NQ cells from 101 deletion-mutant strains were tested for viability and reproductive capacity.  Twenty-one mutants were identified, including 7 that affected reproductive capacity of both cell types.  Thirteen affected only Q or NQ cells, indicating significant differentiation of these cell types.  doa4 strains, lacking ubiquitin hydrolase, affected viability and reproductive capacity in both cell types.  More than 1300 mRNAs differentiating Q and NQ cell fractions were identified by microarray analysis.  Gene-ontology analysis of Q-cell mRNAs showed significant increases in protein-encoding mRNAs involved in membrane maintenance, oxidative stress response, and signal transduction.  NQ-cell mRNAs encode proteins involved in Ty-element transposition and DNA recombination, consistent with apoptosis in these cells.  Consistent with preparation for rapid response to environmental stimuli, approximately 2000 protease-labile mRNAs were identified in Q cells.  The differentiation of these cell types and the ability of genes to selectively affect the survival of Q or NQ cells in yeast are relevant to chronological aging, cell-cycle, genome-evolution, and stem-cell research and provides insight into complex responses that even simple organisms have to starvation.
+  Keywords: Cell-type comparison
+:arrays: 
+  GSM211749: 
+    condition: Non-quiescent
+  GSM211738: 
+    condition: Quiescent
+  GSM211750: 
+    condition: Non-quiescent
+  GSM211739: 
+    condition: Quiescent
+  GSM211751: 
+    condition: Non-quiescent
+  GSM211752: 
+    condition: Non-quiescent
+  GSM211730: 
+    condition: Quiescent
+  GSM211753: 
+    condition: Non-quiescent
+  GSM211731: 
+    condition: Quiescent
+  GSM211754: 
+    condition: Non-quiescent
+  GSM211732: 
+    condition: Quiescent
+  GSM211755: 
+    condition: Non-quiescent
+  GSM211733: 
+    condition: Quiescent
+  GSM211734: 
+    condition: Quiescent
+  GSM211746: 
+    condition: Non-quiescent
+  GSM211735: 
+    condition: Quiescent
+  GSM211747: 
+    condition: Non-quiescent
+  GSM211736: 
+    condition: Quiescent
+  GSM211748: 
+    condition: Non-quiescent
+  GSM211737: 
+    condition: Quiescent
+:title: BY4742 Quiescent and Non-quiescent replicates
+:platform: GPL5627

data/install_scripts/GEO/series/GSE8558.yaml

@@ -0,0 +1,19 @@
+--- 
+:description: |-
+  Cells in glucose-limited Saccharomyces cerevisiae cultures differentiate into quiescent (Q) and non-quiescent (NQ) fractions prior to entering stationary phase.  To identify genes involved in this differentiation, Q and NQ cells from 101 deletion-mutant strains were tested for viability and reproductive capacity.  Twenty-one mutants were identified, including 7 that affected reproductive capacity of both cell types.  Thirteen affected only Q or NQ cells, indicating significant differentiation of these cell types.  doa4 strains, lacking ubiquitin hydrolase, affected viability and reproductive capacity in both cell types.  More than 1300 mRNAs differentiating Q and NQ cell fractions were identified by microarray analysis.  Gene-ontology analysis of Q-cell mRNAs showed significant increases in protein-encoding mRNAs involved in membrane maintenance, oxidative stress response, and signal transduction.  NQ-cell mRNAs encode proteins involved in Ty-element transposition and DNA recombination, consistent with apoptosis in these cells.  Consistent with preparation for rapid response to environmental stimuli, approximately 2000 protease-labile mRNAs were identified in Q cells.  The differentiation of these cell types and the ability of genes to selectively affect the survival of Q or NQ cells in yeast are relevant to chronological aging, cell-cycle, genome-evolution, and stem-cell research and provides insight into complex responses that even simple organisms have to starvation.
+  Keywords: cell type comparison
+:arrays: 
+  GSM211882: 
+    condition: Non-Quiescent PK
+  GSM211883: 
+    condition: Non-Quiescent Tris
+  GSM211884: 
+    condition: Quiescent PK
+  GSM211885: 
+    condition: Quiescent Tris
+  GSM211886: 
+    condition: StationaryPhase PK
+  GSM211887: 
+    condition: StationaryPhase Tris
+:title: Proteinase K treatment of Q and NQ cells
+:platform: GPL5626

data/install_scripts/GEO/series/GSE8559.yaml

@@ -0,0 +1,47 @@
+--- 
+:description: |-
+  Cells in glucose-limited Saccharomyces cerevisiae cultures differentiate into quiescent (Q) and non-quiescent (NQ) fractions prior to entering stationary phase.  To identify genes involved in this differentiation, Q and NQ cells from 101 deletion-mutant strains were tested for viability and reproductive capacity.  Twenty-one mutants were identified, including 7 that affected reproductive capacity of both cell types.  Thirteen affected only Q or NQ cells, indicating significant differentiation of these cell types.  doa4 strains, lacking ubiquitin hydrolase, affected viability and reproductive capacity in both cell types.  More than 1300 mRNAs differentiating Q and NQ cell fractions were identified by microarray analysis.  Gene-ontology analysis of Q-cell mRNAs showed significant increases in protein-encoding mRNAs involved in membrane maintenance, oxidative stress response, and signal transduction.  NQ-cell mRNAs encode proteins involved in Ty-element transposition and DNA recombination, consistent with apoptosis in these cells.  Consistent with preparation for rapid response to environmental stimuli, approximately 2000 protease-labile mRNAs were identified in Q cells.  The differentiation of these cell types and the ability of genes to selectively affect the survival of Q or NQ cells in yeast are relevant to chronological aging, cell-cycle, genome-evolution, and stem-cell research and provides insight into complex responses that even simple organisms have to starvation.
+  Keywords: cell type comparison
+:arrays: 
+  GSM211771: 
+    condition: S288C Non-quiescent
+  GSM211760: 
+    condition: S288C Quiescent
+  GSM211772: 
+    condition: S288C Non-quiescent
+  GSM211761: 
+    condition: S288C Quiescent
+  GSM211773: 
+    condition: S288C Non-quiescent
+  GSM211762: 
+    condition: S288C Quiescent
+  GSM211774: 
+    condition: S288C Non-quiescent
+  GSM211763: 
+    condition: S288C Quiescent
+  GSM211775: 
+    condition: S288C Non-quiescent
+  GSM211764: 
+    condition: S288C Quiescent
+  GSM211765: 
+    condition: S288C Quiescent
+  GSM211766: 
+    condition: S288C Non-quiescent
+  GSM211767: 
+    condition: S288C Non-quiescent
+  GSM211756: 
+    condition: S288C Quiescent
+  GSM211768: 
+    condition: S288C Non-quiescent
+  GSM211757: 
+    condition: S288C Quiescent
+  GSM211770: 
+    condition: S288C Non-quiescent
+  GSM211769: 
+    condition: S288C Non-quiescent
+  GSM211758: 
+    condition: S288C Quiescent
+  GSM211759: 
+    condition: S288C Quiescent
+:title: S288c Quiescent and Non-quiescent replicates
+:platform: GPL5626

data/install_scripts/GEO/series/GSE8613.yaml

@@ -0,0 +1,19 @@
+--- 
+:description: |-
+  Expression microarray experiments were performed to identify all of the aerobic and hypoxic transcripts in wild-type cells.  The role of Hap1 in the regulation of transcription was examined by monitoring gene expression in hap1 deletion cells.
+  Keywords: gene expression, strain comparison, response to hypoxic conditions
+:arrays: 
+  GSM213541: 
+    condition: Aerobic/hypoxic hap1
+  GSM213542: 
+    condition: Aerobic/hypoxic hap1
+  GSM213461: 
+    condition: Aerobic/hypoxic wt
+  GSM213538: 
+    condition: Aerobic/hypoxic wt
+  GSM213527: 
+    condition: Aerobic/hypoxic wt
+  GSM213540: 
+    condition: Aerobic/hypoxic hap1
+:title: Role of S. cerevisiae Hap1 in oxygen-regulation
+:platform: GPL4625

data/install_scripts/GEO/series/GSE8629.yaml

@@ -0,0 +1,18 @@
+--- 
+:description: "Time course after the addition of the transcriptional inhibitor thiolutin at 3 and 10 \xC2\xB5g/mL to an exponential growing culture of S. cerevisiae in YPD.\n\
+  Keywords: time course, mRNA stability analysis"
+:arrays: 
+  GSM213750: 
+    condition: thiolutin_t0min
+  GSM213749: 
+    condition: thiolutin_3ug t30min
+  GSM175046: 
+    condition: thiolutin_t0min
+  GSM175049: 
+    condition: thiolutin_3ug t30min
+  GSM213748: 
+    condition: "thiolutin_10\xC2\xB5g_t30min"
+  GSM175051: 
+    condition: "thiolutin_10\xC2\xB5g_t30min"
+:title: "Yeast mRNA decay analysis after addition of 3 & 10 \xC2\xB5/mL thiolutin"
+:platform: GPL3763

data/install_scripts/GEO/series/GSE8729.yaml

@@ -0,0 +1,19 @@
+--- 
+:title: Overexpression of transcriptional factors Kin28 and Pog1 suppresses the stress sensitivity caused by the rsp5 mutation
+:arrays: 
+  GSM216550: 
+    time: 60min
+    overexpression: KIN28 overexp
+  GSM216549: 
+    time: 0min
+    overexpression: KIN28 overexp
+  GSM216551: 
+    time: 0min
+    overexpression: POG1 overexp
+  GSM216552: 
+    time: 60min
+    overexpression: POG1 overexp
+:description: |-
+  Rsp5 is an essential and multi-functional E3 ubiquitin ligase in Saccharomyces cerevisiae. We previously isolated the Ala401Glu rsp5 mutant, which is hypersensitive to various stresses. To understand the function of Rsp5 in stress responses, suppressor genes whose overexpression allows rsp5A401E cells to grow at high temperature were screened. The KIN28 and POG1 genes, encoding a subunit of the transcription factor TFIIH and a putative transcriptional activator, respectively, were identified as multicopy suppressors of not only high temperature but also LiCl stresses. The overexpression of Kin28 and Pog1 in rsp5A401E cells caused an increase in the transcriptional level of some stress proteins when exposed to temperature up-shift. DNA microarray analysis under LiCl stress revealed that the transcriptional level of some proteasome components was increased in rsp5A401E cells overexpressing Kin28 or Pog1. These results suggest that the overexpression of Kin28 and Pog1 enhances the protein refolding and degradation pathways in rsp5A401E cells.
+  Keywords: mutant analysis, stress response
+:platform: GPL90

data/install_scripts/GEO/series/GSE8761.yaml

@@ -0,0 +1,55 @@
+--- 
+:description: |-
+  Duplicated genes escape gene loss by conferring a dosage benefit or evolving diverged functions.  The yeast Saccharomyces cerevisiae contains many duplicated genes encoding ribosomal proteins.  Prior studies have suggested that these duplicated proteins are functionally redundant and affect cellular processes in proportion to their expression.  In contrast, through studies of ASH1 mRNA in yeast, we demonstrate paralog-specific requirements for the translation of localized mRNAs.  Intriguingly, these paralog-specific effects are limited to a distinct subset of duplicated ribosomal proteins.  Moreover, transcriptional and phenotypic profiling of cells lacking specific ribosomal proteins reveals differences between the functional roles of ribosomal protein paralogs that extend beyond effects on mRNA localization.  Finally, we show that ribosomal protein paralogs exhibit differential requirements for assembly and localization. Together, our data indicate complex specialization of ribosomal proteins for specific cellular processes, and support the existence of a ribosomal code.
+  Keywords: genetic modification
+:arrays: 
+  GSM217633: 
+    condition: Rpl7b
+  GSM217634: 
+    condition: Rpl12a
+  GSM217745: 
+    condition: Rps0a
+  GSM217635: 
+    condition: Rpl12a
+  GSM217746: 
+    condition: Rps0a
+  GSM217636: 
+    condition: Rpl12b
+  GSM217395: 
+    condition: wt
+  GSM217747: 
+    condition: Rps0b
+  GSM217637: 
+    condition: Rpl12b
+  GSM217748: 
+    condition: Rps0b
+  GSM217638: 
+    condition: Rpl14a
+  GSM217616: 
+    condition: wt
+  GSM217750: 
+    condition: Rps18a
+  GSM217749: 
+    condition: Rps18a
+  GSM217640: 
+    condition: Rpl22a
+  GSM217639: 
+    condition: Rpl14a
+  GSM217617: 
+    condition: Rpl7a
+  GSM217751: 
+    condition: Rps18b
+  GSM217641: 
+    condition: Rpl22a
+  GSM217618: 
+    condition: Rpl7a
+  GSM217752: 
+    condition: Rps18b
+  GSM217642: 
+    condition: Rpl22b
+  GSM217643: 
+    condition: Rpl22b
+  GSM217621: 
+    condition: Rpl7b
+:title: Transcriptional profiling of ribosomal protein knockouts
+:platform: GPL90

data/install_scripts/GEO/series/GSE8765.yaml

@@ -0,0 +1,15 @@
+--- 
+:description: |-
+  Duplicated genes escape gene loss by conferring a dosage benefit or evolving diverged functions. The yeast Saccharomyces cerevisiae contains many duplicated genes encoding ribosomal proteins. Prior studies have suggested that these duplicated proteins are functionally redundant and affect cellular processes in proportion to their expression. In contrast, through studies of ASH1 mRNA in yeast, we demonstrate paralog-specific requirements for the translation of localized mRNAs. Intriguingly, these paralog-specific effects are limited to a distinct subset of duplicated ribosomal proteins. Moreover, transcriptional and phenotypic profiling of cells lacking specific ribosomal proteins reveals differences between the functional roles of ribosomal protein paralogs that extend beyond effects on mRNA localization. Finally, we show that ribosomal protein paralogs exhibit differential requirements for assembly and localization. Together, our data indicate complex specialization of ribosomal proteins for specific cellular processes, and support the existence of a ribosomal code.
+  Keywords: genetic modification
+:arrays: 
+  GSM217790: 
+    condition: Loc1
+  GSM217792: 
+    condition: Loc1
+  GSM217793: 
+    condition: Loc1
+  GSM217796: 
+    condition: Loc1
+:title: Transcriptional profiling of Loc1 knockout
+:platform: GPL5741

data/install_scripts/GEO/series/GSE8805.yaml

@@ -0,0 +1,64 @@
+--- 
+:arrays: 
+  GSM218326: 
+    condition: Ras*tpkw gal 0min vs. 40min
+  GSM218315: 
+    condition: WT gal 0min vs. 20min
+  GSM218327: 
+    condition: Ras* gal 0min vs. 60min
+  GSM218316: 
+    condition: WT gal 0min vs. 40min
+  GSM218328: 
+    condition: Ras*tpkw gal 0min vs. 60min
+  GSM218317: 
+    condition: WT gal 0min vs. 20min
+  GSM218306: 
+    condition: tpk-w gal 0min vs. 60min
+  GSM218330: 
+    condition: Ras*tpkw gal 0min vs. 60min
+  GSM218329: 
+    condition: Ras* gal 0min vs. 60min
+  GSM218318: 
+    condition: WT gal 0min vs. 40min
+  GSM218307: 
+    condition: tpk-w gal 0min vs. tpk-w gal 40min
+  GSM218331: 
+    condition: WT glu 0min vs. 60min
+  GSM218320: 
+    condition: Ras* gal 0min vs. 40min
+  GSM218319: 
+    condition: Ras* gal 0min vs. 20min
+  GSM218308: 
+    condition: WT gal 0min vs. WT gal 20min
+  GSM218332: 
+    condition: WT gal 0min vs. 60min
+  GSM218321: 
+    condition: Ras* gal 0min vs. 20min
+  GSM218310: 
+    condition: tpk-w gal 0min vs. 40min
+  GSM218309: 
+    condition: tpk-w gal 0min vs. 20min
+  GSM218333: 
+    condition: WT glu 0min vs. 60min
+  GSM218322: 
+    condition: Ras* gal 0min vs. 40min
+  GSM218311: 
+    condition: WT glu 0min vs. 20min
+  GSM218334: 
+    condition: WT gal 0min vs. 60min
+  GSM218323: 
+    condition: Ras*tpkw gal 0min vs. 40min
+  GSM218312: 
+    condition: WT glu 0min vs. 40min
+  GSM218324: 
+    condition: Ras*tpkw gal 0min vs. 20min
+  GSM218313: 
+    condition: WT glu 0min vs. 20min
+  GSM218325: 
+    condition: Ras*tpkw gal 0min vs. 20min
+  GSM218314: 
+    condition: WT glu 0min vs. 40min
+:description: "To assess the relative contributions of the PKA and MAPK pathways in Ras stimulation of filamentation, we examined the change in transcriptional profile following activation of Ras in strains in which one of these two pathways has been severed. These arrays contain the experiments involving WT strain with glucose addition, Ras2G19V strain with galactose induction, tpk2-w strain as a control, and RasG19V activation in the tpk2-w background. Activation of Ras in the \xCE\xA31278b background \xE2\x80\x93 by galactose addition to a gal1 PGAL10-RAS2G19V strain - causes a massive restructuring of the transcription pattern of the cell, which closely resembles the changes induced by addition of glucose. To assess the extent to which these Ras-induced changes depended on signaling through PKA, we compared the expression profile following induction of activated Ras in a wild type background to that following induction of activated Ras in the bcy1 tpk1 tpk3 tpk2V218G.\n\
+  Keywords: time course"
+:title: Ras Affects Filamentous Growth in Saccharomyces cerevisiae Solely Through Regulation of Protein Kinase A
+:platform: GPL884

data/install_scripts/GEO/series/GSE8825.yaml

@@ -0,0 +1,79 @@
+--- 
+:description: |-
+  We studied the relation between growth rate and genomewide gene expression, cell cycle progression, and glucose metabolism in 36 steady state continuous cultures limited by one of six different nutrients (glucose, ammonium, sulfate, phosphate, uracil or leucine).  The expression of more than a quarter of all yeast genes is linearly correlated with growth rate, independently of the limiting nutrient.  The subset of negatively growth-correlated genes is most enriched for peroxisomal functions, whereas positively correlated genes mainly encode ribosomal functions.  Many (not all) genes associated with stress response are strongly correlated with growth rate, as are genes that are periodically expressed under conditions of metabolic cycling.  We confirmed a linear relationship between growth rate and the fraction of the cell population in the G0/G1 cell cycle phase, independent of limiting nutrient.  Cultures limited by auxotrophic requirements wasted excess glucose, whereas those limited on phosphate, sulfate or ammonia did not; this phenomenon (reminiscent of the "Warburg effect" in cancer cells) was confirmed in batch cultures.  Using an aggregate of gene expression values, we predict (in both continuous and batch cultures) an "instantaneous growth rate".  This concept is useful in interpreting the systemlevel connections among growth rate, metabolism, stress and the cell cycle.
+  Keywords: growth condition design
+:arrays: 
+  GSM219294: 
+    condition: LlimD.2
+  GSM219283: 
+    condition: PlimD.25
+  GSM219272: 
+    condition: ClimD.3
+  GSM219295: 
+    condition: LlimD.25
+  GSM219284: 
+    condition: PlimD.3
+  GSM219273: 
+    condition: NlimD.05
+  GSM219296: 
+    condition: LlimD.3
+  GSM219285: 
+    condition: SlimD.05
+  GSM219274: 
+    condition: NlimD.1
+  GSM219297: 
+    condition: UlimD.05
+  GSM219286: 
+    condition: SlimD.1
+  GSM219275: 
+    condition: NlimD.15
+  GSM219298: 
+    condition: UlimD.1
+  GSM219287: 
+    condition: SlimD.15
+  GSM219276: 
+    condition: NlimD.2
+  GSM219299: 
+    condition: UlimD.15
+  GSM219288: 
+    condition: SlimD.2
+  GSM219277: 
+    condition: NlimD.25
+  GSM219300: 
+    condition: UlimD.2
+  GSM219289: 
+    condition: SlimD.25
+  GSM219278: 
+    condition: NlimD.3
+  GSM219267: 
+    condition: ClimD.05
+  GSM219301: 
+    condition: UlimD.25
+  GSM219290: 
+    condition: SlimD.3
+  GSM219279: 
+    condition: PlimD.05
+  GSM219268: 
+    condition: ClimD.1
+  GSM219302: 
+    condition: UlimD.3
+  GSM219291: 
+    condition: LlimD.05
+  GSM219280: 
+    condition: PlimD.1
+  GSM219269: 
+    condition: ClimD.15
+  GSM219292: 
+    condition: LlimD.1
+  GSM219281: 
+    condition: PlimD.15
+  GSM219270: 
+    condition: ClimD.2
+  GSM219293: 
+    condition: LlimD.15
+  GSM219282: 
+    condition: PlimD.2
+  GSM219271: 
+    condition: ClimD.25
+:title: Coordination of Growth Rate, Cell Cycle, Stress Response and Metabolic Activity in Yeast
+:platform: GPL884